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Motes HIV-1 replication in infected cells by antagonizing the effects of the interferon (IFN) pathway. In vitro studies have shown that Tat could inhibit IFN mediated nitric oxide synthase activity, which is essential, otherwise, to containment of pathogens by macrophages (Barton et al., 1996). Tat has also been reported to induce the expression of the suppressor of cytokine signaling? (SOCS-2), w
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